Which condition is not typically caused by organophosphate poisoning?

Organophosphate poisoning is primarily associated with disruptions in the nervous system due to the inhibition of acetylcholinesterase, an enzyme essential for the breakdown of the neurotransmitter acetylcholine. This leads to a buildup of acetylcholine, causing overstimulation of both the central and peripheral nervous systems. The symptoms that arise from this poisoning typically include severe constriction of pupils, inability to walk due to neuromuscular effects, and various respiratory and cardiovascular symptoms, which can include mild chest discomfort.

Increased appetite, however, is not a symptom associated with organophosphate poisoning. Instead, the physiological response to organophosphate exposure generally leads to symptoms such as nausea, vomiting, and abdominal cramping, all of which would likely reduce a person's appetite rather than increase it. This highlights that option referring to increased appetite is not aligned with the expected clinical manifestations of organophosphate poisoning, making it the correct choice in this scenario.